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Here, we demonstrate the critical nature of mitochondrial dynamics in MM cells and suggest how these machineries could be potentially exploited by upregulating CD38 surface levels: we assume that the huge intracellular NAD + depletion triggered by CD38 upregulation unmasks mitochondria machinery dysfunction, thus making it an actionable vulnerability for MM cells

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Today, the compound continues to be referenced in scientific literature involving peptide chemistry, molecular signalling and laboratory-based biological research