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Dysregulated NFE2L2 expression impairs exercise performance, energy expenditure, and mitochondrial function, underscoring the importance of this pathway for muscle health and adaptation to physical activity [15]

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Moreover, recent experimental results show that two more histidine residues located in the C terminal region of PrP provide protection against neurotoxicity, when this is exerted from the N terminal region (Schilling et al., 2020)