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Summary and perspectives This review highlights the pivotal role of amino acid metabolism in the pathogenesis of pulmonary fibrosis, with a focus on how dysregulated metabolic pathways—particularly those involving proline, arginine, glutamine, and tryptophan—drive collagen deposition, fibroblast activation, and disease progression
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Glutathione has been involved in many different studies, including those on patients with Parkinson’s disease, cancer, Alzheimer’s, and others
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