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Deletion of GPRASP2 causes impaired synaptic communications and modulates the surface availability of mGluR5 in mice [61]
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Additionally, NAC supplementation downregulated the expression of pro-inflammatory cytokines (TNF-α, IL-1β, and IL-6) and upregulated genes associated with aging (such as sirtuin-1) and neuroprotection (neuroglobin, synapsin-I, and MBP-2)
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