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491 Based on inactivation studies and shunt metabolite analysis, this enzyme was initially predicted to catalyze chlorination of an unactivated sp 3 carbon in orthosporin ( 266 ) to yield demethyldichlorodiaporthin ( 267 ) (Figure 56)
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Fe 2+ leaves the intestinal epithelial cells through the pathway mediated by FPN1 when the intracellular iron is too high (14)
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