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For instance, CRAMP, a cathelicidin identified in mouse, can affect the survival and replication of the influenza A virus (IAV) in host cells by directly disrupting the IAV envelope (Gallo et al., LL-37, a peptide derived from the human cathelicidin, has been shown to resist HSV-1 infection (Lee et al., 2019), whereas LL-37 is limited in its therapeutic use due to its length of 37 residues, resulting in high chemical synthesis costs

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