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The mechanisms by which this occurs are indirect via regulation of hormonal signaling pathways such as prolactin and GnRH, acting as a cofactor in enzymatic processes that affects testosterone regulation and production, modulating of androgen receptor sensitivity, protecting against oxidative stress, and reducing homocysteine toxicity (Fig
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These findings were not surprising and could be explained by the fact that there was an increase in the amount of unsaturated fatty acids in poorly differentiated tumours
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