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The Successful Treatment of a Patient with Ehlers–Danlos Syndrome (EDS) After an Extensive Burn Injury: A Case Report

Indeed, recent studies of clinical cases and animal models have revealed a number of physiological alterations (excitatory/inhibitory (E/I) neurotransmitter imbalance in the hippocampus [24], altered working memory and other functions associated with dorsolateral prefrontal cortex (DLPFC) [25], impaired sleep [26], etc.), appearing in response to a deficit of GABAergic interneuron-dependent inhibition [27], loss [28] or excessive production of GABAergic neurons [29], or compromised function of the main components of the GABA pathway (Figure 2), which are the focus of this paper

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