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Our analyses demonstrated that PD patients exhibit significant metabolic reprogramming, characterized by a shift in energy metabolism from the tricarboxylic acid cycle toward glycolysis, a dysregulated urea cycle, and lipid remodeling, as well as extensive activation of inflammatory and immune responses involving the PI3K-Akt, IL-17, NF-kappaB, MAPK and TNF signaling pathways

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[23] BCR-ABL transformation is associated with genomic instability, so that most patients who receive continuous treatment using all-trans retinoic acid and imatinib mesylate are easy to develop disease resistance