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The latter acts as a catalyst in redox reactions and lipid peroxidation, resulting in TBARS as the final product (Pandey and Rizvi, 2010)
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Here, as an additional perspective to overviews associating excessive ROS production with “oxidative stress” as previously put forward [1,2], we concentrate on the necessity for ROS-S systems, with a focus upon some of their crucial roles in normal endocrine cardio-metabolic physiology
It also plays a key role in the immune system
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