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The exact mechanism for Dabrafenib’s penetration across the BLB has not been revealed, but its unique otoprotective property is conditional on its translocation into the cochlea to counteract cisplatin-induced BRAF signaling cascade and hair cell death (Ingersoll et al., 2020)

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In contrast, tumor cells remain highly susceptible to high-dose radiation, and evidence suggests that the efficacy of FLASH-RT in treating tumors may be comparable to or even surpass conventional radiotherapy [203–205]