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home > autism glutathione genes spectrum disorder at the crossroad between and environment: contributions, convergences, and interactions in ASD developmental pathophysiology | Molecular Autism Honest review on easy > autism glutathione genes spectrum disorder at the crossroad between and environment: contributions, convergences, and interactions in ASD developmental pathophysiology | Molecular Autism Honest review on easy
autism glutathione genes spectrum disorder at the crossroad between and environment: contributions, convergences, and interactions in ASD developmental pathophysiology | Molecular Autism Honest review on easy
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autism glutathione genes spectrum disorder at the crossroad between and environment: contributions, convergences, and interactions in ASD developmental pathophysiology | Molecular Autism Honest review on easy
autism glutathione genes spectrum disorder at the crossroad between and environment: contributions, convergences, and interactions in ASD developmental pathophysiology | Molecular Autism Honest review on easy
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autism glutathione genes spectrum disorder at the crossroad between and environment: contributions, convergences, and interactions in ASD developmental pathophysiology | Molecular Autism Honest review on easy
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autism glutathione genes spectrum disorder at the crossroad between and environment: contributions, convergences, and interactions in ASD developmental pathophysiology | Molecular Autism Honest review on easy

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In a previous study, mitochondrial fusion protein-2 (MFN2) enhanced mitochondria-endoplasmic reticulum (ER) contact and fostered inflow of mitochondrial Ca 2+ required for efficient mitochondrial metabolism by interacting with the ER-embedded Ca-ATPase SERCA2, thereby preventing mitochondrial Ca 2+ overaccumulation and apoptosis

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autism glutathione genes spectrum disorder at the crossroad between and environment: contributions, convergences, and interactions in ASD developmental pathophysiology | Molecular Autism Honest review on easy

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