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Animal studies have determined that excessive stimulation of the 5HT2A receptor is primarily responsible for SS, 4 and that 5HT2A antagonists, such as mirtazapine, can block the development of SS in a mouse coadministered 5 In addition to a patient’s medication history, the clinical hallmark of SS that helps distinguish it from neuroleptic malignant syndrome, delirium, and other acute syndromes is clonus, which becomes spontaneous and sustained as the severity increases

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