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Because IGFBP sequestration is the primary mechanism controlling IGF-1 bioavailability and clearance, removing this regulatory layer dramatically extends the circulating half-life to approximately 20-30 hours compared to 10-15 minutes for native IGF-1
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These studies suggest a close relationship between copper and necroptosis, indicating that copper can serve as a therapeutic target for inducing necroptosis in tumor cells and has potential application prospects in tumor treatment
Clinically proven
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